Abstract
We report on a pyogenic psoas abscess secondary to an impacted calcium oxalate ureteric stone in a 2-year-old boy with glycogen storage disease type 1 (GSD-1). The patient had a drainage of the abscess through a flank incision followed by percutaneous nephrostomy and open ureterolithotomy. Metabolic acidosis, hyperuricemia, hypocitraturia, and hypercalciuria appear to be significant in the pathogenesis of urolithiasis in patients with GSD-1. Regular ultrasonography of the abdomen along with optimal metabolic control may delay or prevent urolithiasis and its complications in GSD-1 patients.
Original language | English |
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Pages (from-to) | 1772-1775 |
Number of pages | 4 |
Journal | Pediatric Nephrology |
Volume | 21 |
Issue number | 11 |
DOIs | |
Publication status | Published - Nov 2006 |
Keywords
- Glycogen storage disease
- Nephrocalcinosis
- Psoas abscess
- Urolithiasis